Alcohol is a toxin and it can have mild to severe effects on our body, depending on the severity of abuse. In mild cases, alcohol can cause lower inhibitions, concentration troubles, loss of coordination, reduce core body temperature, cause vomiting, and even causing sufferers to pass out. Your contribution helps us support research and provide an improved quality of life for those affected. Thus, further preclinical and clinical studies are required to assess of this molecule in alcoholic neuropathy.
Symptoms and Signs of Alcoholic Neuropathy
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Supplementing with vitamin B12, folate, vitamin alcohol neuropathy E, and thiamine may be advised to address these deficits. Alcoholic neuropathy is suggested by certain patterns, including slowing of nerve activity, reduced amplitude of nerve waves, and diminished function in the hands and feet. Only the degree of nerve damage is determined by the nerve tests; the cause of neuropathy is not. Heat sensitivity, unusual feelings like “pins and needles,” and numbness or painful sensations in the arms and legs are common signs of sensory problems. Individuals’ level of pain is determined by how severe their polyneuropathy is.
- The reason for better results in the benfotiamine alone group than in the Milgamma-N group, despite the fact that the benfotiamine dosage was equivalent, is not completely understood.
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- Light contact can feel unpleasant and exaggerated, especially in the fingers and toes.
- Research suggests you can recover from some or all of the nerve damage caused by alcohol-related neuropathy.
Treatment / Management
Thus, these vitamin deficiencies were not considered to be major causal factors of neuropathy 26. In one clinical study, aimed at studying distinct clinicopathologic features of alcoholic neuropathy, 64 patients were assessed. In 47 of these patients sural nerve biopsy was performed, with discrimination in terms of their thiamine status 3. The ethanol consumption of these patients was more than 100 g day–1 for more than 10 years. The subgroup without thiamine deficiency consisted of 36 patients, while the subgroup with thiamine deficiency consisted of 28 patients. In addition, 32 patients with nonalcoholic thiamine deficiency neuropathy were also evaluated for comparison.
Nutritional factors responsible for alcoholic neuropathy (indirect toxicity)
- The toxic effects of alcohol may damage your peripheral nerves, which play a role in movement and sensation.
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- A doctor may diagnose a person with alcoholic neuropathy, if alcohol use has damaged the peripheral nerves.
- Alcohol decreases the absorption of nutrients such as magnesium, selenium, and vitamins B1 and B2, causing significant deficits that affect many areas of the body, including the nerves.
- Alcoholic neuropathy is one of the most common adverse effects of chronic alcohol consumption.
The sooner you stop drinking alcohol, the more favorable your outlook is if you have alcohol-related neuropathy. Research suggests you can recover from some or all of the nerve damage caused by alcohol-related neuropathy. The medical community has recognized that addiction is a disease and some people are predisposed to it.
Outlook of alcoholic neuropathy
About 46% of all chronic alcohol users may eventually develop this condition. It is likely to get worse if the person continues to use alcohol or if nutritional problems are not corrected. Alcoholic neuropathy is usually not life threatening, but it can severely affect quality of life.
It’s estimated that anywhere between 25 to 66 percent of people with an AUD have some form of alcoholic neuropathy. People with an AUD drink frequently, and if they don’t seek treatment, the disorder can be long-lasting. Reversing the condition includes having a good, healthy diet full of supplements and nutrients to replace the lost nutrients over the years. It is recommended to eat foods that are high in nutritional value, and not too Substance abuse heavy on the body’s metabolism. However, if caught early enough, you can minimize the damage from alcoholic neuropathy.
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